Publikationen in Zusammenarbeit mit Forschern von University of Freiburg (16)

2016

  1. Sox9 and Sox8 protect the adult testis from male-to-female genetic reprogramming and complete degeneration

    eLife, Vol. 5, Núm. JUN2016

2012

  1. Genes promoting and disturbing testis development

    Histology and Histopathology, Vol. 27, Núm. 11, pp. 1361-1383

  2. L-Sox5 and Sox6 proteins enhance chondrogenic miR-140 MicroRNA expression by strengthening dimeric Sox9 activity

    Journal of Biological Chemistry, Vol. 287, Núm. 26, pp. 22206-22215

  3. Sox9 and Sox8 are required for basal lamina integrity of testis cords and for suppression of FOXL2 during embryonic testis development in mice

    Biology of Reproduction, Vol. 87, Núm. 4

2010

  1. Dicer is required for Sertoli cell function and survival

    International Journal of Developmental Biology, Vol. 54, Núm. 5, pp. 867-875

  2. Profiling spermatogenic failure in adult testes bearing Sox9-deficient Sertoli cells identifies genes involved in feminization, inflammation and stress

    Reproductive Biology and Endocrinology, Vol. 8

  3. Role of apoptosis and cell proliferation in the testicular dynamics of seasonal breeding mammals: A study in the Iberian mole, Talpa occidentalis

    Biology of Reproduction, Vol. 83, Núm. 1, pp. 83-91

  4. SOX E genes: SOX9 and SOX8 in mammalian testis development

    International Journal of Biochemistry and Cell Biology

2009

  1. Testis cord differentiation after the sex determination stage is independent of Sox9 but fails in the combined absence of Sox9 and Sox8

    Developmental Biology, Vol. 327, Núm. 2, pp. 301-312

  2. The PGD2 pathway, independently of FGF9, amplifies SOX9 activity in Sertoli cells during male sexual differentiation

    Development, Vol. 136, Núm. 11, pp. 1813-1821

2008

  1. Mutations in the cyclin family member FAM58A cause an X-linked dominant disorder characterized by syndactyly, telecanthus and anogenital and renal malformations

    Nature Genetics, Vol. 40, Núm. 3, pp. 287-289

  2. Sall1, Sall2, and Sall4 are required for neural tube closure in mice

    American Journal of Pathology, Vol. 173, Núm. 5, pp. 1455-1463

  3. Sox9 is required for invagination of the otic placode in mice

    Developmental Biology, Vol. 317, Núm. 1, pp. 213-224

2006

  1. Homozygous inactivation of Sox9 causes complete XY sex reversal in mice

    Biology of Reproduction, Vol. 74, Núm. 1, pp. 195-201

  2. Long-range upstream and downstream enhancers control distinct subsets of the complex spatiotemporal Sox9 expression pattern

    Developmental Biology, Vol. 291, Núm. 2, pp. 382-397

  3. Sox9 is required for notochord maintenance in mice

    Developmental Biology, Vol. 295, Núm. 1, pp. 128-140