Publicaciones en colaboración con investigadores/as de Centro de Investigaciones Biológicas (18)

2021

  1. Identification of exosomal microrna signature by liquid biopsy in hereditary hemorrhagic telangiectasia patients

    International Journal of Molecular Sciences, Vol. 22, Núm. 17

2019

  1. MMP-12, secreted by pro-inflammatory macrophages, targets endoglin in human macrophages and endothelial cells

    International Journal of Molecular Sciences, Vol. 20, Núm. 12

2018

  1. Human endoglin as a potential new partner involved in platelet–endothelium interactions

    Cellular and Molecular Life Sciences, Vol. 75, Núm. 7, pp. 1269-1284

2016

  1. Mice Lacking Endoglin in Macrophages Show an Impaired Immune Response

    PLoS Genetics, Vol. 12, Núm. 3

  2. Transcription factor KLF6 upregulates expression of metalloprotease MMP14 and subsequent release of soluble endoglin during vascular injury

    Angiogenesis, Vol. 19, Núm. 2, pp. 155-171

2014

  1. Expression of endoglin isoforms in the myeloid lineage and their role during aging and macrophage polarization

    Journal of Cell Science, Vol. 127, Núm. 12, pp. 2723-2735

2013

  1. Endothelial endoglin is involved in inflammation: Role in leukocyte adhesion and transmigration

    Blood, Vol. 121, Núm. 2, pp. 403-415

2012

  1. Oxysterol-induced soluble endoglin release and its involvement in hypertension

    Circulation, Vol. 126, Núm. 22, pp. 2612-2624

  2. Structural and functional insights into endoglin ligand recognition and binding

    PLoS ONE, Vol. 7, Núm. 2

2011

  1. Activin A skews macrophage polarization by promoting a proinflammatory phenotype and inhibiting the acquisition of anti-inflammatory macrophage markers

    Blood, Vol. 117, Núm. 19, pp. 5092-5101

2010

  1. Involvement of the TGF-β superfamily signalling pathway in hereditary haemorrhagic telangiectasia

    Journal of Applied Biomedicine, Vol. 8, Núm. 3, pp. 169-177

  2. SETBP1 overexpression is a novel leukemogenic mechanism that predicts adverse outcome in elderly patients with acute myeloid leukemia

    Blood, Vol. 115, Núm. 3, pp. 615-625

2008

  1. Caveolin-1 interacts and cooperates with the transforming growth factor-β type I receptor ALK1 in endothelial caveolae

    Cardiovascular Research, Vol. 77, Núm. 4, pp. 791-799

  2. Mutation study of Spanish patients with Hereditary Hemorrhagic Telangiectasia

    BMC Medical Genetics, Vol. 9

  3. S-endoglin expression is induced in senescent endothelial cells and contributes to vascular pathology

    Circulation Research, Vol. 103, Núm. 12, pp. 1383-1392

2007

  1. Structural Model of Human Endoglin, a Transmembrane Receptor Responsible for Hereditary Hemorrhagic Telangiectasia

    Journal of Molecular Biology, Vol. 365, Núm. 3, pp. 694-705

2006

  1. Hereditary hemorrhagic telangiectasia, a vascular dysplasia affecting the TGF-β signaling pathway

    Clinical Medicine and Research, Vol. 4, Núm. 1, pp. 66-78

2005

  1. Interaction and functional interplay between endoglin and ALK-1, two components of the endothelial transforming growth factor-β receptor complex

    Journal of Cellular Physiology, Vol. 204, Núm. 2, pp. 574-584