LUIS CARLOS LÓPEZ GARCÍA-rekin lankidetzan egindako argitalpenak (65)

2022

  1. Melatonin drives apoptosis in head and neck cancer by increasing mitochondrial ROS generated via reverse electron transport

    Journal of Pineal Research, Vol. 73, Núm. 3

  2. The Q-junction and the inflammatory response are critical pathological and therapeutic factors in CoQ deficiency

    Redox Biology, Vol. 55

2020

  1. Coenzyme Q10 modulates sulfide metabolism and links the mitochondrial respiratory chain to pathways associated to one carbon metabolism

    Human Molecular Genetics, Vol. 29, Núm. 19, pp. 3296-3311

2019

  1. β-RA reduces DMQ/CoQ ratio and rescues the encephalopathic phenotype in Coq9 R239X mice

    EMBO Molecular Medicine, Vol. 11, Núm. 1

  2. Lack of NLRP3 Inflammasome Activation Reduces Age-Dependent Sarcopenia and Mitochondrial Dysfunction, Favoring the Prophylactic Effect of Melatonin

    Journals of Gerontology - Series A Biological Sciences and Medical Sciences, Vol. 74, Núm. 11, pp. 1699-1708

  3. Melatonin enhances cisplatin and radiation cytotoxicity in head and neck squamous cell carcinoma by stimulating mitochondrial ROS generation, apoptosis, and autophagy

    Oxidative Medicine and Cellular Longevity, Vol. 2019

  4. Rapamycin administration is not a valid therapeutic strategy for every case of mitochondrial disease

    EBioMedicine, Vol. 42, pp. 511-523

2018

  1. Combination of melatonin and rapamycin for head and neck cancer therapy: Suppression of AKT/mTOR pathway activation, and activation of mitophagy and apoptosis via mitochondrial function regulation

    Journal of Pineal Research, Vol. 64, Núm. 3

  2. In Vivo Determination of Mitochondrial Respiration in 1-Methyl-4-Phenyl-1,2,3,6-Tetrahydropyridine-Treated Zebrafish Reveals the Efficacy of Melatonin in Restoring Mitochondrial Normalcy

    Zebrafish, Vol. 15, Núm. 1, pp. 15-26

  3. Reduction in the levels of CoQ biosynthetic proteins is related to an increase in lifespan without evidence of hepatic mitohormesis

    Scientific Reports, Vol. 8, Núm. 1

  4. The Protective Effect of Melatonin Against Age-Associated, Sarcopenia-Dependent Tubular Aggregate Formation, Lactate Depletion, and Mitochondrial Changes

    Journals of Gerontology - Series A Biological Sciences and Medical Sciences, Vol. 73, Núm. 10, pp. 1330-1338

2017

  1. CoQ deficiency causes disruption of mitochondrial sulfide oxidation, a new pathomechanism associated with this syndrome

    EMBO Molecular Medicine, Vol. 9, Núm. 1, pp. 78-95

  2. Melatonin administration to wild-type mice and nontreated NLRP3 mutant mice share similar inhibition of the inflammatory response during sepsis

    Journal of Pineal Research, Vol. 63, Núm. 1

  3. Melatonin enhances neural stem cell differentiation and engraftment by increasing mitochondrial function

    Journal of Pineal Research, Vol. 63, Núm. 2

  4. Melatonin protects rats from radiotherapyinduced small intestine toxicity

    PLoS ONE, Vol. 12, Núm. 4

  5. Melatonin, clock genes and mitochondria in sepsis

    Cellular and Molecular Life Sciences, Vol. 74, Núm. 21, pp. 3965-3987

  6. Mitochondrial impairment and melatonin protection in parkinsonian mice do not depend of inducible or neuronal nitric oxide synthases

    PLoS ONE, Vol. 12, Núm. 8

2016

  1. Identification of morphological markers of sarcopenia at early stage of aging in skeletal muscle of mice

    Experimental Gerontology, Vol. 83, pp. 22-30

  2. Melatonin rescues zebrafish embryos from the parkinsonian phenotype restoring the parkin/PINK1/DJ-1/MUL1 network

    Journal of Pineal Research, pp. 96-107

  3. Permeabilized myocardial fibers as model to detect mitochondrial dysfunction during sepsis and melatonin effects without disruption of mitochondrial network

    Mitochondrion, Vol. 27, pp. 56-63