Publikationen, an denen er mitarbeitet ROSARIO JIMÉNEZ MOLEÓN (31)

2016

  1. Vascular and central activation of peroxisome proliferator-activated receptor-β attenuates angiotensin II-induced hypertension: Role of RGS-5

    Journal of Pharmacology and Experimental Therapeutics, Vol. 358, Núm. 1, pp. 151-163

2015

  1. Antihypertensive effects of probiotics Lactobacillus strains in spontaneously hypertensive rats

    Molecular Nutrition and Food Research, Vol. 59, Núm. 11, pp. 2326-2336

  2. Chronic peroxisome proliferator-activated receptorβ/δ agonist GW0742 prevents hypertension, vascular inflammatory and oxidative status, and endothelial dysfunction in diet-induced obesity

    Journal of Hypertension, Vol. 33, Núm. 9, pp. 1831-1844

  3. Plan de acción tutorial biorienta2. Orientación a futuros farmacéuticos: La investigación en biomedicina como carrera profesional. Tercera edición (PID 12-70)

    Innovación docente y buenas prácticas en la Universidad de Granada. (Editorial Universidad de Granada), pp. 520-531

2014

  1. Chronic hydroxychloroquine improves endothelial dysfunction and protects kidney in a mouse model of systemic lupus erythematosus

    Hypertension, Vol. 64, Núm. 2, pp. 330-337

  2. PPARβ activation restores the high glucose-induced impairment of insulin signalling in endothelial cells

    British Journal of Pharmacology, Vol. 171, Núm. 12, pp. 3089-3102

2013

  1. Effects of peroxisome proliferator-activated receptor-β activation in endothelin-dependent hypertension

    Cardiovascular Research, Vol. 99, Núm. 4, pp. 622-631

  2. SIRT1 inhibits NADPH oxidase activation and protects endothelial function in the rat aorta: Implications for vascular aging

    Biochemical Pharmacology, Vol. 85, Núm. 9, pp. 1288-1296

2012

  1. Activation of peroxisome proliferator-activated receptor-β/-δ (PPARβ/δ) prevents endothelial dysfunction in type 1 diabetic rats

    Free Radical Biology and Medicine, Vol. 53, Núm. 4, pp. 730-741

  2. Different cardiovascular protective effects of quercetin administered orally or intraperitoneally in spontaneously hypertensive rats

    Food and Function, Vol. 3, Núm. 6, pp. 643-650

  3. Epicatechin lowers blood pressure, restores endothelial function, and decreases oxidative stress and endothelin-1 and NADPH oxidase activity in DOCA-salt hypertension

    Free Radical Biology and Medicine, Vol. 52, Núm. 1, pp. 70-79

2011

  1. Antihypertensive effects of peroxisome proliferator-activated receptor-β activation in spontaneously hypertensive rats

    Hypertension, Vol. 58, Núm. 4, pp. 733-743

  2. BIOrienta2.orientación a futuros farmacéuticos: La Investigación en biomedicina como carrera profesional

    Univest 2011: III Congreso Internacional "La autogestión del aprendizaje" (Universitat de Girona = Universidad de Gerona)

  3. Chronic (-)-epicatechin improves vascular oxidative and inflammatory status but not hypertension in chronic nitric oxide-deficient rats

    British Journal of Nutrition, Vol. 106, Núm. 9, pp. 1337-1348

  4. Red wine polyphenols prevent endothelial dysfunction induced by endothelin-1 in rat aorta: Role of NADPH oxidase

    Clinical Science, Vol. 120, Núm. 8, pp. 321-333

  5. Vascular deconjugation of quercetin glucuronide: The flavonoid paradox revealed?

    Molecular Nutrition and Food Research, Vol. 55, Núm. 12, pp. 1780-1790

2010

  1. Endothelium-dependent vasodilator effects of peroxisome proliferator-activated receptor β agonists via the phosphatidyl-inositol-3 kinase-Akt pathway

    Journal of Pharmacology and Experimental Therapeutics, Vol. 332, Núm. 2, pp. 554-561

  2. Lack of beneficial metabolic effects of quercetin in adult spontaneously hypertensive rats

    European Journal of Pharmacology, Vol. 627, Núm. 1-3, pp. 242-250

  3. Vascular superoxide production by endothelin-1 requires Src non-receptor protein tyrosine kinase and MAPK activation

    Atherosclerosis, Vol. 212, Núm. 1, pp. 78-85

2009

  1. Quercetin inhibits vascular superoxide production induced by endothelin-1: Role of NADPH oxidase, uncoupled eNOS and PKC

    Atherosclerosis, Vol. 202, Núm. 1, pp. 58-67