Publicaciones en colaboración con investigadores/as de University of the Witwatersrand (18)

2022

  1. Clinical practice in giant cell arteritis based on a survey of specialists

    Revista Clinica Espanola, Vol. 222, Núm. 5, pp. 266-271

  2. IgA Vasculitis: Influence of CD40, BLK and BANK1 Gene Polymorphisms

    Journal of Clinical Medicine, Vol. 11, Núm. 19

2021

  1. Author Correction: BAFF, APRIL and BAFFR on the pathogenesis of Immunoglobulin-A vasculitis (Scientific Reports, (2021), 11, 1, (11510), 10.1038/s41598-021-91055-z)

    Scientific Reports

  2. BAFF, APRIL and BAFFR on the pathogenesis of Immunoglobulin-A vasculitis

    Scientific Reports, Vol. 11, Núm. 1

  3. Cranial and extracranial giant cell arteritis do not have different HLA-DRB1 and HLA-B association in Caucasian individuals

    Arthritis Research and Therapy

  4. HLA association with the susceptibility to anti-synthetase syndrome

    Joint Bone Spine, Vol. 88, Núm. 3

  5. Role of MUC1 rs4072037 polymorphism and serum KL-6 levels in patients with antisynthetase syndrome

    Scientific Reports, Vol. 11, Núm. 1

  6. Role of the IL33 and IL1RL1 pathway in the pathogenesis of Immunoglobulin A vasculitis

    Scientific Reports, Vol. 11, Núm. 1

2020

  1. Association Between Homocysteine Serum Level and Bone Mineral Density in Patients With Rheumatoid Arthritis

    Journal of Clinical Densitometry, Vol. 23, Núm. 4, pp. 561-567

  2. Association between homocysteine levels and arterial stiffness in women with systemic sclerosis

    Clinical and experimental rheumatology, Vol. 38, Núm. 3, pp. 92-97

  3. Influence of IL17A gene on the pathogenesis of immunoglobulin-A vasculitis

    Clinical and experimental rheumatology, Vol. 38, Núm. 2, pp. 166-170

  4. Influence of MUC5B gene on antisynthetase syndrome

    Scientific Reports, Vol. 10, Núm. 1

  5. Role of IRF5 in the pathogenesis of immunoglobulin-A vasculitis

    Clinical and experimental rheumatology, Vol. 38, Núm. 2, pp. 182-187

2019

  1. Giant cell arteritis: Is the clinical spectrum of the disease changing?

    BMC Geriatrics, Vol. 19, Núm. 1

2017

  1. A genome-wide association study suggests the HLA Class II region as the major susceptibility locus for IgA vasculitis /692/699/1670 /692/4023/1670/595 article

    Scientific Reports, Vol. 7, Núm. 1

2016

  1. Interleukin 1 beta (IL1ß) rs16944 genetic variant as a genetic marker of severe renal manifestations and renal sequelae in Henoch-Schönlein purpura

    Clinical and Experimental Rheumatology, Vol. 34, pp. S84-S88

2015

  1. Brief report: Association of HLA-DRB101 with IgA vasculitis (henoch-schönlein)

    Arthritis and Rheumatology, Vol. 67, Núm. 3, pp. 823-827

  2. Role of PTPN22 and CSK gene polymorphisms as predictors of susceptibility and clinical heterogeneity in patients with Henoch-Schönlein purpura (IgA vasculitis)

    Arthritis Research and Therapy, Vol. 17, Núm. 1